He was also taking two medications known to be associated with pancreatitis: dipeptidyl peptidase-4 [15] and a sodium-glucose cotransporter-2 inhibitor [16], which could have worsened or triggered his symptoms given the background of undiagnosed hypertriglyceridemia. Although his hypertriglyceridemia was likely long-standing, the normal enzyme levels were less likely due to acute and chronic disease as he never had prior symptoms and there was no evidence of calcification on imaging. This evidence concerns the gene SLC5A2 and hypertriglyceridemia.