TP53 and viral myocarditis: Conversely, the NonMet-BC group exhibited significantly enriched pathways, such as apoptosis, xenobiotic biodegradation, and metabolism (drug metabolism—cytochrome P450, fluorobenzoate degradation, 1,1,1-Trichloro-2,2-bis(4-chlorophenyl)ethane (DDT) degradation, chlorocyclohexane and chlorobenzene degradation, and styrene degradation), pathways related to bacterial and viral infections (influenza A, pathogenic E. coli infection, toxoplasmosis, viral myocarditis), non-homologous end-joining, and the tumor suppressor p53 signaling pathways (Figure 6).