IL17A and metabolic dysfunction-associated steatotic liver disease: In a high-fat diet (HFD)-induced NAFLD mouse model, IL-17A and IL17-RA expression increased after HFD challenge; in comparison, the knockout of IL17-RA increased steatosis but decreased steatohepatitis, suggesting a central role of the IL-17 axis in the development and progression of NAFLD to steatohepatitis [73].