Moreover, other factors indirectly related to inflammation, such as functional iron deficiency and anemia [23], have been purported to affect FGF23 transcription and cleavage from the full intact molecule (iFGF23) into its fragments, such as the C-terminal end of the FGF23 molecule (cFGF23), that is involved in other biological actions and active in different biological pathways [16,23]. This evidence concerns the gene FGF23 and nutritional disorder.