In addition to macrophages, carbamylated HDL is also reported to inhibit SR-BI signaling in human aortic endothelial cells (HAECs), accompanied by a suppressed expression of vascular endothelial growth factor receptor-2 (VEGFR2) and impaired endothelial repair capabilities, which can together contribute to endothelial dysfunction [142]. Here, KDR is linked to endothelial dysfunction.