The promoter regions of IFI16, SLC1A2, and SLC1A3 were largely unmethylated in astrocytes both in controls and patients with autism (Table 3) but were moderately methylated in neurons (signifying neuronal identity vs. astrocytes) where IFI16 and SLC1A3 were hypomethylated (~50%, and 40%, respectively, Table 3 and Figure 2H,I) and SLC1A2 exhibited no change in autism (Table 3). The gene discussed is SLC1A3; the disease is autism.