FXR1 is elevated in G0 and chemosurviving acute monocytic leukemia (AML) cells, and this resistance-phenotype seems to be driven by a variety of ribosomal alterations, one of them being an increase in the incorporation of the ribosome stalk protein uL10 (RPLP0). The gene discussed is RPLP0; the disease is acute myeloid leukemia.