While inflammation normallyacts as a protective mechanism, it can cause damage to thebody when it becomes uncontrollable (Dinarello, 2018).IL-1 has been implicated in neuronal cell damage (Allanet al., 2005), and excessive phagocytosis may contribute topathologies in Alzheimer’s disease, schizophrenia, and aging(Vilalta, Brown, 2018). This evidence concerns the gene IL1A and Alzheimer disease.