SOAT1 and cyst: The data showed that the expression levels of esg‐lacZ in somatic cyst cells were also significantly elevated upon activation of JAK/STAT signalling by hopTum‐l overexpression, a constitutively active form of JAK83; on the contrary, the levels of esg‐lacZ in CySCs were diminished upon STAT depletion (Figure 1D–G).