Consistent with Fujita et al.27, we also observed colocalization of the genetic signal driving CR1 expression in oligodendrocytes with AD GWAS risk signal35 within the Banner cohort (posterior probability: 0.985), with rs679515 emerging as the most likely causal variant explaining this shared signal (posterior probability: 0.817, Fig. 3B, Supplementary Dataset 4). The gene discussed is CR1; the disease is Alzheimer disease.