Consistent with previous research, the overexpression of AXL, a member of the TAM (Tyro3, Axl, and Mer) receptor tyrosine kinase family, in mesenchymal carcinoma cells likely plays a role in the cell-intrinsic immune escape mechanism in NSCLC patients by suppressing the killing effects of NK cells and cytotoxic T lymphocytes (CTLs). This evidence concerns the gene AXL and non-small cell lung carcinoma.