In a screening of BEN2908’s transposon (STM) mutants, we reported that a treA null mutant was reduced in production of type 1 fimbriae, demonstrated decreased adherence and invasion of avian fibroblasts and decreased bladder colonization in the murine model of urinary tract infection (UTI) (Pavanelo et al., 2018). The gene discussed is TREH; the disease is urinary tract infection.