The expression levels of hepatic FGL1 were significantly elevated in the condition of acute liver injury, such as partial hepatectomy and radiation-induced liver injury (25, 41), and in the extrahepatic induction of inflammation, such as subcutaneous injection of a known enhancer of IL-6, turpentine oil, and rheumatoid arthritis (42, 43). The gene discussed is FGL1; the disease is rheumatoid arthritis.