According to previous reports, G3BP1‐mediated formation of SGs or SG‐like structures can activate downstream signalling, including the Wnt/β‐catenin signalling pathway, to facilitate cancer progression.[27] We then investigated whether DCAF7 affects the activation of Wnt/β‐catenin signalling and found that overexpression of DCAF7 enhanced the activation of this pathway. Here, G3BP1 is linked to cancer.