Recently, the oncogenic role of G3BP1 has been implicated in various cancers.[32] For instance, in hepatocellular carcinoma, G3BP1 promotes tumor metastasis by upregulating Slug expression.[33] Conversely, depletion of G3BP1 inhibits PI3K/AKT and Wnt/β‐catenin signalling‐mediated proliferation and metastasis of oesophageal cancer cells.[21b] Additionally, acquisition of the senescence‐associated secretory phenotype mediated by G3BP1 is a major contributor to tumor growth associated with senescence.[34] However, the role of G3BP1 in NPC remains largely unexplored. Here, AKT1 is linked to nasopharyngeal carcinoma.