To validate whether KAT6A LLPS was induced by a gap in the KAT6A expression level (Figure 2), the same amount of KAT6A was overexpressed in primary or PARPi‐resistant ovarian cancer cells, and KAT6A LLPS and the interaction of KAT6A with PARP1 were tested (Figure 6A). This evidence concerns the gene PARP1 and ovarian carcinoma.