However, both KAT6AIDR‐FUS, whose LLPS was rescued by IDR from FUS, and KAT6AC543G/G657E enhanced PARPi resistance in ovarian cancer cells (Figures 1H–J and 4A,B), indicating that KAT6A LLPS, but not its catalytic function, counteracted the effects of PARPi. The gene discussed is FUS; the disease is ovarian carcinoma.