Under normal conditions, TGFβ1 acts as a tumor suppressor and pro‐apoptotic agent in adult hepatocytes, regulating liver mass, while genetic deletion of TGFB1 in mice on a BALB/c background led to the development of necroinflammatory IFN‐γ‐dependent hepatitis.30 Here, TGFB1 is linked to hepatitis A virus infection.