On the other hand, inhibiting PSTNTac1 somas counters the anorectic effect of a malaise-inducing agent (lipopolysaccharide), neophobia (first-time exposure to sucrose), or appetite-suppressing hormones (amylin, cholecystokinin, peptide YY), while ablating PSTNVGluT2 neurons abolishes anorexia induced by glucagon-like peptide-1 [12, 14, 19] and silencing PSTN neurons as a whole (CaMKIIα promoter) attenuates cholecystokinin-induced and fear-induced feeding suppression [18, 20]. Here, CCK is linked to Anorexia.