PABPC1 and gastric cancer: reported a novel methylation‐independent function of METTL3 in the cytoplasm, where METTL3 promotes gastric cancer by upregulating mRNA translation of specific transcripts.[55] METTL3 interacts with PABPC1, stabilizing its association with the cap‐binding complex eIF4F, preferentially enhancing the translation of epigenetic factors without m6A modifications.