reported a novel methylation‐independent function of METTL3 in the cytoplasm, where METTL3 promotes gastric cancer by upregulating mRNA translation of specific transcripts.[55] METTL3 interacts with PABPC1, stabilizing its association with the cap‐binding complex eIF4F, preferentially enhancing the translation of epigenetic factors without m6A modifications. This evidence concerns the gene EIF4G1 and gastric cancer.