Studies have shown that type 2 diabetes mellitus (T2DM) is a pro-inflammatory state with the evidence of increased levels of high-sensitivity C-reactive protein (hs-CRP) and pro-inflammatory cytokines: interleukin 1β (IL-1β), interleukin 6 (IL-6) and tumour necrosis factor alpha (TNF-α), among others (Misra, Das & Sahu 2012). This evidence concerns the gene IL1B and type 2 diabetes mellitus.