Subsequent molecular-genetic testing for the most common autosomal-recessive form of HL (DFNB1A, OMIM: 220290) confirmed that the contribution of causative variants of the GJB2 gene to the etiology of HL in Buryat patients was only 5.1% (one of the lowest rates in the world), while in Russian patients the contribution was 28.9%18, which was comparable with the results of segregation analysis17. Here, GJB2 is linked to Hodgkins lymphoma.