ATP7B and Wilson disease: Further, EMFs from fresh and frozen murine liver tissue and from frozen liver of a rat model for Wilson disease (diseased Atp7b−/− knockout and healthy Atp7b+/− control rats), a genetic disease causing hepatic, and in consequence mitochondrial Cu accumulation [11] were compared regarding Mn, Fe, and Cu content, as a proof of concept for the assessment of quantitative TE differences.