Additionally, HULC functioned as a competitive endogenous RNA (ceRNA) of miR-3200-5p and inhibited ferroptosis by targeting ATF4, leading to the promotion of cell proliferation and metastasis in HCC, suggesting the potential for targeting HULC/miR-3200-5p/ATF4 axis in HCC treatment [129] (Fig. 2). The gene discussed is ATF4; the disease is hepatocellular carcinoma.