We subsequently hypothesized that since CT‐1 was downregulated in NTS nephritis and CT‐1 treatment has been shown to improve other models of kidney disease induced by unilateral ureteral obstruction (Perretta‐Tejedor et al., 2019), ischemia (Garcia‐Cenador et al., 2013) and toxins (Quiros et al., 2016), it may also have a protective effect in glomerulonephritis. This evidence concerns the gene CTF1 and kidney disorder.