In line with this, the overexpression of SARS‐CoV‐2‐encoded non‐structural protein 1 (NSP1), which binds to the 40S ribosomal subunits and inhibits general mRNA translation [100], significantly alleviates neuromuscular degeneration caused by ribosome collision in models of Alzheimer's disease (AD), Parkinson's disease (PD), and amyotrophic lateral sclerosis (ALS) [101]. The gene discussed is SH2D3A; the disease is Alzheimer disease.