In another study using human fetal neuroepithelial cells (hNSCs) obtained from the endbrain of human fetuses aborted at 13 weeks and implanted in AD mice, TrkA/B phosphorylation in AD mice was significantly increased, which led to increased phosphorylation levels of AKT and GSK3β(Ser9). The gene discussed is AKT1; the disease is Alzheimer disease.