MTOR and Epstein-Barr virus infection: More importantly, KEGG analysis indicated that a plethora of signaling pathways involved in CGN were significantly enriched, such as immune system-related Th1 and Th2 cell differentiation, Th17 cell differentiation, and T cell receptor signaling pathway; antiviral response-related human T-cell leukemia virus 1 infection, Epstein-Barr virus infection, and human immunodeficiency virus 1 infection; inflammation response-related Notch signaling pathway, PPAR signaling pathway, and mTOR signaling pathway (Figure 6(E)).