Finally, we sought to determine whether glaucoma led to disruption in the size and/or number of presynaptic GABAergic terminals in the dLGN, as changes to the presynaptic vGAT have been implicated in activity-dependent presynaptic homeostatic plasticity (De Gois et al., 2005; Prestigio et al., 2021; Fig. 6). Here, SLC32A1 is linked to glaucoma.