Among neurologic tumors, in neuroblastoma (NB), lncRNA MEG3 functions as an anti-tumor agent, and overexpressing MEG3 in NB cells decreased epithelial-mesenchymal transition invasion and metastasis via mTOR signaling and inhibited FOXO1-mediated autophagy (Ye et al., 2020). This evidence concerns the gene MTOR and neuroblastoma.