Prior research has implicated the involvement of HOXA10 in epithelial‐mesenchymal transition (EMT), a critical process in cancer metastasis.[67, 68] In light of the significant changes in metastatic phenotypes following HOXA10 expression alteration observed in our study (Figure 6f,g), we sought to determine if these metastatic phenotypes are associated with EMT initiation driven by HOXA10 in PCa cells. The gene discussed is HOXA10; the disease is posterior cortical atrophy.