Although this result can be explained based on the selected FASTA sequence of the TRIM33 gene, which was restricted to the coding portion, complementation of multiple lncRNAs at TRIM33 intron 1 may reflect interference with canonical and alternative splicing, perhaps reducing the expression of functional mRNAs or generating alternatively spliced isoforms of TIF1γ that would promote autoimmunity. Here, TRIM33 is linked to Autoimmunity.