Through the examination of GWAS data, noteworthy correlations have been unveiled between the SNPs situated within the DNA repair enzyme EEPD1, and changes in metabolic traits (including NAFLD and hyperlipidemia) as well as hypoxia‐related traits (including reticulocyte count and CT70), all of which are considered as consequence of OSAS. Here, CT70 is linked to metabolic dysfunction-associated steatotic liver disease.