The prevalent clinical-pathological manifestations observed in CanL encompass mild to moderate normocytic normochromic and non-regenerative anemia, lymphopenia, hyperproteinemia characterized by elevated gamma-globulins and beta-globulins, along with hypoalbuminemia, leading to a reduced albumin/globulin ratio and proteinuria [9,34]. This evidence concerns the gene ALB and Hypoalbuminemia.