REN and Nephropathy: Upon reviewing the literature, the etiology of renal injury in ACS patients is intricate, arising from a combination of factors such as hemodynamic abnormalities due to reduced cardiac output and venous return, activation of the sympathetic nervous system, and the renin–angiotensin–aldosterone system (RAAS), leading to vasoconstriction and exacerbating kidney damage, disruptions in the coagulation system, activation of the inflammatory response, and oxidative stress [37,38,39,40].