Additionally, in the particular setting of CS-AKI, Radovic et al. evaluated NGAL (Neutrophil Gelatinase-Associated Lipocalin), KIM-1 (Kidney Injury Molecule-1), and sLac levels in CS patients assessed as low-risk for developing CS-AKI, and they found out that postoperative sLac was a better predictor of CS-AKI than NGAL and KIM-1 [28]. This evidence concerns the gene HAVCR1 and Cowden syndrome 1.