Lastres-Becker et al. [18] showed that sporadic and SOD1-ALS-immortalized lymphocytes indicate elevated levels of ROS and increased lipid peroxidation, mainly in sporadic ALS lymphoblasts, which is in accordance with the assumption that oxidative stress plays a key role in the pathogenesis of neurodegenerative diseases, including ALS [18]. This evidence concerns the gene SOD1 and amyotrophic lateral sclerosis.