In CIV neurons, we find that, although high repeat mutant Huntingtin (mHTT) expression results in dendritic hypotrophy and a loss of underlying microtubule signals, like that of TORC1 and CCT LOF, we do not find evidence that mHTT disrupts the TORC1–CCT dendritic arborization pathway in HD conditions. Here, CRTC1 is linked to Huntington disease.