The present study hypothesis focused on the analyzes of seven axes directly associated with Type 2 Diabetes (T2D), including Glut-4 reactivation, improvement in the Insulin Resistant Pathway (DGKd and PKCε), IR activation, activation of insulin secretion through the reactivation of the island of Langerhans EGFR Pathway Inhibition, Reduction of Inflammatory Cytokines (TNF-α, IL-Ib, IL-6), andReduction in Fatty Changes. The gene discussed is PRKCE; the disease is type 2 diabetes mellitus.