ELN and infarction: The physiological and/or pathological degradation of elastin results in release of so-called elastin-derived-peptides (EDPs), which are detected in many tissues, e.g. in infarction-affected brain (measured in cerebrospinal fluid and varying between 6.3 ng/mL to 129.5 ng/mL) and/or in human serum (depending on the tested group, the amount of EDPs varied between 11.9 ng/mL to 81.1 ng/mL) [4, 5].