LINC00987 and acute myeloid leukemia: Furthermore, LINC00987 downregulation reduced the proliferation, clone formation, IC 50, drug resistance proteins (P-GP and BCRP), and the volume of Xenograft tumor where promoted apoptosis under ADR treatment in ADR-resistance AML cell, while LINC00987 overexpression in ADR-sensitive AML cell played the opposite role.