In fact, Gustafsson and peers analyzed and confirmed this potential link as they discovered that patients with type A fibrils develop cardiomyopathy and heart failure (or their preexisting cardiomyopathy deteriorates) after liver transplantation, while the same is not seen in patients with type B fibrils, further suggesting that type A fibrils are more susceptible to continuous amyloid deposition from wild-type TTR [121]. Here, TTR is linked to cardiomyopathy.