NKT cells were the major source of IFNγ in the unstimulated state and continuously expressed IFNγ, while the IFNγ upregulation was detected only in ILCs 1h after Con A treatment (Fig. 2C), indicating that ILCs, rather than NKT cells, were stimulated to produce IFNγ at the early stage of Con A-induced ALI. Here, IFNG is linked to acute respiratory distress syndrome.