In a study by Cakir et al,[21] which focused on inflammation, demyelination, reactive gliosis, and neuronal damage characterizing multiple sclerosis (MS) as a potentially progressive autoimmune disorder of the central nervous system, the relationship between salusin alpha and SAL-β peptides and inflammation, as well as their association with relapsing-remitting MS (RRMS), was investigated. The gene discussed is TOR2A; the disease is relapsing-remitting multiple sclerosis.