A previous study reported that loss of function of the SWI/SNF complex due to mutations in its subunits promotes the progression of thyroid tumors and renders thyroid tumors resistant to redifferentiation therapies.[21] After analyzing the RNA‐seq results, it was observed that the expression of several subunits of the SWI/SNF complex was increased following SETMAR overexpression (Figure 4D). This evidence concerns the gene SMARCA1 and thyroid tumor.