A previous report from Wuhan, China showed that five of the first 41 patients (12%) with COVID-19 had the evidence of myocardial injury, such as elevated high-sensitivity cardiac troponin I (cTnI, > 28 pg/mL) levels (Zheng et al. 2020; Huang et al. 2020; Lippi et al. 2020), and 7.2–17% of COVID-19 inpatients had acute myocardial injury (Wang et al. 2020a; Zhou et al. 2020a). This evidence concerns the gene TNNI3 and injury.