Furthermore, we observed that the independent DEGs of silenced NEAT1 were involved in many cancer pathways, the MAPK pathway, the mTOR pathway, the Wnt pathway and the PI3K/AKT pathway, while the independent DEGs of silenced NEAT1 were only enriched in gastric cancer, breast cancer and hepatocellular carcinoma (Fig. 7G, H). This evidence concerns the gene MTOR and breast carcinoma.