It was observed that specifically in mice with type 2 diabetes mellitus (T2DM) that were treated orally with 200 mg kg−1 and in insulin-resistant cells, C-PC increased insulin sensitivity and the cellular use of glucose through the activation of AMP-activated protein kinase (AMPK), which, once active, promotes autophagy, and of serine/threonine kinase 1 (AKT), which is part of the phosphatidylinositol-3 kinase (PI3K) pathway and is relevant in the regulation of the glyco-metabolism [71]. The gene discussed is INS; the disease is diabetes mellitus.