These changes are closely related to important pathways such as phosphoinositide 3-kinase (PI3K)/ protein kinase B (AKT)/ mechanistic target of rapamycin (mTOR) [61] and sterol regulatory element-binding proteins (SREBPs) [62], which play critical roles in ovarian cancer cell proliferation, apoptosis, and lipid synthesis. This evidence concerns the gene MTOR and ovarian cancer.