In another recent comprehensive study, Canesin et al. [95] determined the impact on the apheresis product arising from mobilization (or lack of) with G-CSF and plerixafor from 30 healthy donors, by assessing immune cell composition, T-cell phenotype, and T-cell functionality in controlling AML tumor growth following anti-CD33 CAR transduction. Here, CSF3 is linked to neoplasm.