Sekine et al. also confirmed the specificity of YAP1-NUTM1 and YAP1-MAML2 (or reciprocal) fusion products for poroma and PC, since these were not found in any other cutaneous tumor (including SCC, BCC, MCC, cutaneous adenocarcinoma and seborrheic keratosis), except for poroid hidradenoma. This evidence concerns the gene MAML2 and seborrheic keratosis.