A study by Haustein and co-workers [34] demonstrated the cannabinoid-induced upregulation of ICAM-1 (intercellular adhesion molecule-1) on lung cancer cells to be responsible for increased cancer cell lysis by LAK (lymphokine-activated killer) cells, thus providing evidence of a novel antitumorigenic mechanism and the properties of CBD. The gene discussed is ICAM1; the disease is lung carcinoma.